ABFRACCIONES DENTALES PDF

el 81% de los pacientes con edades comprendidas entre 45 a 59 años presentaban abfracciones, EDAD se asocia significativamente con las. DENTALES. abfraction la abfracción abrasive elabrasivo abrasion of teeth abscess abutment acid acidulated phosphate fluoride acrylic appliance active caries. Tooth wear or tooth surface loss is a normal physiological process and occurs throughout life but is considered pathological when the degree of.

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The loss by wear of tooth substance or a restoration caused by mastication or contact between occluding or approximal surfaces. This pain is due to the enamel having been eroded away, exposing the sensitive dentin.

As such, clinicians may encounter significant difficulties in determining the dominant aetiology. There is a high consumption of fruits, abfraccionrs juices and carbonated beverages in Trinidad and Tobago and this may be similar to other Caribbean islands. Based on the optical changes induced in eroded tissue by the lesions, in Koshoji et al. There is theoretical evidence to support the concept of abfraction, but little experimental evidence exists.

ABFRACCIONES: LESIONES CERVICALES NO CARIOSAS EN CUÑA, SU RE by Ana Maria De Stefani on Prezi

Upper anterior teeth restored by bonding tooth-coloured composite dental materials. This page was last edited on 13 Julyat As abfraction is still a controversial theory there are various ideas on what causes the lesions.

Tooth wear or tooth surface loss is a normal physiological process and occurs throughout life but is considered pathological when the degree of destruction is excessive or the rate of loss is rapid, causing functional, aesthetic or sensitivity problems.

There are numerous signs of dental erosion, including changes in appearance and sensitivity. In-vivo studies are advantageous in assessing erosion directly from the patient’s mouth. Basal cell adenoma Canalicular adenoma Ductal papilloma Monomorphic adenoma Myoepithelioma Oncocytoma Papillary cystadenoma lymphomatosum Pleomorphic adenoma Sebaceous adenoma Malignant: P 13 March Diagnoses Chronic periodontitis Localized aggressive periodontitis Generalized aggressive periodontitis Periodontitis as a manifestation of systemic disease Necrotizing periodontal diseases Abscesses of the periodontium Combined periodontic-endodontic lesions.

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Do adhesive resins protect dentine from erosion by acids?

3-STEP Technique, a simplified protocol for ADDITIVE adhesive rehabilitations

This occurs because the white enamel has eroded away to reveal the yellowish dentin beneath. More active restorative treatment involving simple restorations Fig. Temporomandibular jointsmuscles of mastication and malocclusions — Jaw joints, chewing muscles and bite abnormalities Bruxism Condylar resorption Mandibular dislocation Malocclusion Crossbite Open bite Overbite Overeruption Overjet Prognathia Retrognathia Scissor bite Maxillary hypoplasia Temporomandibular joint dysfunction.

The findings of a study in Trinidad indicate that the prevalence abfracciknes tooth wear in a Trinidadian population is comparable to the United Kingdom UK and, indeed, that the level of moderate and severe wear is nearly twice as high 2.

Salivary glands Benign lymphoepithelial lesion Ectopic salivary gland tissue Frey’s syndrome HIV salivary gland disease Necrotizing sialometaplasia Mucocele Ranula Pneumoparotitis Salivary duct stricture Salivary gland aplasia Salivary gland atresia Salivary gland diverticulum Salivary gland fistula Salivary gland hyperplasia Salivary gland hypoplasia Salivary gland neoplasms Benign: Particular questions to be asked are type of toothbrush used, whether hard or soft, toothbrushing frequency and history of bruxism grinding or clenching.

Dental erosion is the most common chronic disease of children ages 5—17, [1] although it is only relatively recently that it has been recognised as a dental health problem. Those thought to pose a risk are soft drinks, some alcohol and fruit drinks, fruit juices such as orange juice abbfracciones contain citric acid and carbonated drinks such as colas in which the carbonic acid is not the cause of erosion, but citric and phosphoric acid.

This mainly happens on the first, second, and third molars. Reflux past the upper oesophageal sphincter has been shown to increase the risk for erosion in the mouth 5.

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Dental Erosion and Medical Conditions An Overview of Aetiology, Diagnosis and Management

A change in shape of the teeth is also a sign of dental erosion. L Truelove October A follow-up study of 18 subjects with extensively worn dentitions”. A variety of drinks contribute to dental erosion due to their low pH level. In statements such as these there is no comment on whether the lesions occur above or below the CEJ.

The acid that causes erosive wear may be classified as intrinsic or extrinsic 10 depending on the source of the acid from either the stomach intrinsic or the diet and other environmental sources extrinsic. One of the most prevalent theories is called “the theory of non-carious cervical lesions” which suggests that tooth flexion, occurring due to occlusion factors, impacts on the vulnerable area near the cementoenamel junction.

Saliva acts as a buffer, regulating the pH when acidic drinks are ingested. This page was last edited on 1 Octoberat To provide the best treatment option the dental clinician must determine the level of activity and predict possible progression of the lesion.

Periapical, mandibular and maxillary hard tissues — Bones of jaws. West Indian Med J. Erosion is highly prevalent in people of all ages.

Erosion is the progressive loss of dental hard abfracclones by acid from a non-bacterial source 8. One of the physical changes can be the color of teeth. Stomach acid can also enter the oral cavity during vomiting episodes due to alcohol hangovers, chronic alcoholism, morning sickness associated with pregnancy, eating disorders such as anorexia and bulimia nervosa 11 and with voluntary regurgitation or rumination